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I really have to find a new hobby. Anyway, I'll try to keep this theory short. It is well known that many medications that mitigate (sometimes very effectively) HPPD symptoms, either directly or indirectly inhibit calcium channels. Think Levetiracetam (Keppra), Lamotrigine (Lamictal), Flunarizine, Clonidine.. you get the point. New to Neurology (or Ionic transmission etc.)? Have a quick introduction here, to help you comprehend this theory. Now then.. observe the following abstract: That's right, I was too lazy to type it out. Source "Magnesium can block calcium action" Yep... that's it. Guess that doesn't need further explanation so as to what the possible implication of magnesium might be. Experiences anyone? I'll just chalk this up on my list of research targets for the time being. Ohh one thing though: there seems to be little mention of brain specific activity, and mostly cardiovasculair stuff.. I'm just gonna go ahead and presume that calcium inhibition happens throughout the organism in every structure? Anyone with a little more knowledge on that subject, please feel free to comment. I mean, when I look up CCB's on Wiki, there's little info on brain-specific effects.. Only a short mentioning of anti-convulsant effects. On a side note, I noticed that my dear mother uses Amlodipine, which happens to be a calcium channel blocker... Had I known that earlier I might've experimented with that, but with treatment around the corner it seems pointless. WAIT! There's more. For just 9,99... ok sorry. Ahum. Check out this study. My work here is done