yosoydiego
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Posts posted by yosoydiego
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My simple advise: read about Deja Vú.
Science has some understanding on how they happen now. Perhaps once you understand the mechanism you'll feel more at ease with it.
Imagine that your knee was failing you, and you were limping. You WON'T assign that to God dragging your leg or anything like that, right? Well, with this Deja Vu is the same, just that way more complex because it's inside of our mind, but once you interpret it as just a recurrent simple fail of your memory, and take it as knee pain, I think you'll make some progress
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Excellent! Congratulations! I agree with the strong HPPD-stress correlation. I don't buy the Grof's BS though
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Hi, have a look at this, just in case:
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7 hours ago, josht9210 said:
I read that it can be dopamine related and helped with Tolcapone and Levocarb
Where?
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21 hours ago, josht9210 said:
This was very sad to read
Thought exactly the same.
I hope he's getting some help
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Thanks for sharing, glad you're better, keep us posted on the keppra!
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On 10/27/2019 at 3:59 PM, LilDoke said:
getting biophoton therapy 4 months in. and I started to feel that I was getting better. The night after the first biophoton therapy, I woke in something that was like peaking again on LSD. It was clear to me that my body was cleaning out and returning to baseline. Both of these therapies helped a lot and I still do it.
I'd definitely want to hear more about that!
How many sessions did you get? Did you get those after effects every time?
It's extremely rare, so please, tell us all you can!
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2 minutes ago, hope1 said:
I agree. If no research is done which is very much the case now then no breakthroughs are to be expected. www.neurogroup.org is going to fix this problem...
Research doesn't work that way, is not that straightforward...
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22 hours ago, AlphaBeta said:
I'm quite sure that there will never be the one solution, the one etiology, the one medication that cures most people. At least not in my lifetime.
At the rate that technology and medicine advances, I wouldn't say that unless I was 60+ years old....
I don't know how old you are, of course
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23 hours ago, AlphaBeta said:
Sleep got much better meanwhile, but Visuals are still getting worse. My psychiatrist offered me Lamotrigine now because he thinks we should somehow "interrupt" the worsening. I'm quite unsure if I should start a medical treatment at this point. On the one hand, my HPPD is overall still mild, but on the other hand it constantly gets worse. Initially I wanted to wait for at least 8 months before trying medications, but now I'm not so sure anymore. Maybe my doctor is right and I should stop just watching it becoming (may irreversibly) worse. Will think about it within the next few weeks.
If anyone has thoughts or input for me regarding this decision, please let me know.
If it was me, I'd take it asap. It just makes all sense, I don't see any reason to delay treatment. In fact for me it would make sense to do exactly the opposite: get as much treatment asap, when maybe the "damage" is still reversible... and taper off later on.
Netflix documentary, The Mind Explained, episode 5, minute 18, they make the analogy of our brains being like mountains covered in snow, with sleds creating paths on it, once you repeat something long enough, making it difficult to escape that route.
If you let your visuals "sink in", it might be impossible to get out of there.
Just crazy thoughts, of course.
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Felicia, I linked you drugs because you were looking for a "cure for HPPD", and those are the most reliable.
That pursuit, mixed with your eagerness to find information, but yet your lack of skills to properly understand, interpret, and filter what you read, could be a really dangerous mix. Be careful, you're prone to fall into any type of dangerous shit...
"sometimes longer" means a couple of hours more, not days, months, years, etc.
Autophagy is a constant natural process. You could slightly stimulate it, but that's about it.
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Honestly, if you only have OEV, I'd just try to accept them, ignore them, etc.
It seems like the general opinion is that that is the best. Stressing trying to find a "cure" that doesn't exists, could be way worse, stress is the worst. And adding more medications with its side effects, also worse.
But that's just a bold opinion
Best of luck anyway!
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"Stuck" for 12hs. Nothing to do with hppd.
I hope you're referring to "autophagy" as the process that happens naturally in our bodies, and not to actually doing it consciously on a macro level...
Some anecdotes have reported benefits with more or less extreme diets or exercise... but anecdotes.
if you want to give it a try, go ahead, but keep it SAFE!Some supplements that might help: https://www.reddit.com/r/HPPD/comments/aqzv06/lions_manenaccholinemagnesium/
Some prescription drugs that might help: http://hppdonline.com/topic/6012-most-commonly-used-medications/
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On 6/13/2019 at 3:08 PM, facesofhppd.com said:
he had a low count for testosterone
Could you tell us exactly the value?
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Both Haloperidol and risperidone have worked for some people, although be aware that many report some worsening of symptoms at the beginning of the treatment
I guess Keppra helped with DP/DR, but not with open eye visuals?
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"up to 12 hours"
LSD is always fully cleared from your system, although it might take slightly longer than for most other drugs, but it has nothing to do with hppd..
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17 hours ago, dasitmane said:
Also if anyone has had any EEGs done please do post the results.
EEGs are, more or less, useless. Fun though
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10 minutes ago, dasitmane said:
Also, there are arguements that Risperidone, an inverse agonist of 5HTP2A exasperates symptoms of HPPD, which is also probably very true, as per the drug in itself without history of HPPD can cause palinopsia. I'm guessing the difference is just in the dose, as the one guy listed his dose which was very smart of him, and as far as I'm concerned is a pretty low dose, not the lowest, but pretty low, as the high does is 200mg.
Treatment with the atypical antipsychotic medications paliperidone and risperidone normalized basal extracellular glutamate
https://www.ncbi.nlm.nih.gov/pubmed/21699956
In line with my other post
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I think that you're lucky to enjoy your hppd, and to have the photoshop skills to show it to the rest!
If you turn around, not facing the sun, I guess they are gone? In that case, it would be shadows.
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18 hours ago, dasitmane said:
Whats your reason? Theres a decent amount of evidence that its the case at hand. They can prove that it definitely is what causes hallucinations, and antipsychotics that act on the receptor make HPPD symptoms worse in a lot of cases.
To keep it more ordered, I've created a whole topic replying to that:
http://hppdonline.com/topic/6791-hppd-is-not-serotonergic-in-nature/
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I should add the prefix to the subject: "Why I think that", but it would be too long. Consider it added. I'll elaborate some points and quote some sources, but note that many more sources can be found, the conclusion is not based on these sources only.
Note that I'll make a conceptual leap: since basically nothing can be said about HPPD due to lack of research and data, I'll treat it as some form of psychosis/schizophrenia, which is widely studied (although not understood anyway), and REMARKABLY similar to HPPD. At the very least, it could be said that HPPD is a mild form of psychosis/schizophrenia, a subset, etc, but it seems very logical to assume that they will share many characteristics. See second link, about Drug Induced Psychosis
- The causes of schizophrenia are not understood, and many hypothesis have been proposed, notably: serotonergic causes, dopaminergic causes, and glutamate causes.
While the sertonergic idea is the older, newer studies point in other directions, mainly of stimulatory nature
https://link.springer.com/chapter/10.1007/978-1-4684-8228-7_17
- Recent studies have indicated the importance of the Dopaminergic system in the effects of LSD, distinguishing between phases of the LSD effects according to the main receptors activated, and proposing as a model for schizophrenia.
https://www.ncbi.nlm.nih.gov/m/pubmed/21352832
https://www.ncbi.nlm.nih.gov/pubmed/15723230
- While sertonergic system is mainly associated with mood (depression, anti-depressants), the Glutamate system is more related with the information processing and memory formation.
- The relationship between dopamine and glutamate is deep, and many studies are studying their link to each other, and to psychosis.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6162342/
https://www.ncbi.nlm.nih.gov/pubmed/24524997
https://www.nature.com/articles/s41398-017-0071-9
- Going back to David Nichols study about the 2 different phases (sertonergic and dopaminergic) phases of LSD, and comparing that to the description of HPPD, seems clear that HPPD seems more "phase 2". It's usually describe as "the last part of an LSD trip"
- For drugs or situations that are well known to trigger or worsen HPPD according to the community, it can be found that they act on the dopamine/glutamate system, but NOT (or barely) on the sertonergic system.
-- coffee-- stress
-- cocaine
- The inverse argument can be seen also: drugs that act directly on the serotonergic system, usually have little effect on HPPD, or make it even worse. Example: anti-depressants (SSRI). Also, the same drugs are not known to cause HPPD.
- Of the drugs that have consistently shown usefulness for the HPPD community, 2 of the 4 target the Glutamate system
https://www.reddit.com/r/HPPD/comments/aqzv06/lions_manenaccholinemagnesium/
- Some HPPD sufferers are experimenting with Ketamine, with good results. Ketamine's main method of action is through the Glutamate system, not serotonin.
- Regarding stress being a trigger for HPPD, the relationship between cortisol and glutamate and PTSD has been already treated, in this example, by no less than David Nutt, one of the most world renowned scientists regarding drugs:
https://www.ncbi.nlm.nih.gov/pubmed/18701640
- More about the relationship of psychedelics, anti-psychotics, and 5HT2A and Glutamate receptors can be seen here:
- More on the relationship of excess glutamate in the PFC and schizophrenia:In sum, for all of the above, I believe it's pretty clear that HPPD has way bigger chances of being glutamate related, than serotonin related.
I could also keep going on a bit more about in the hypothesis, but I'll leave that for a follow up text.
Looking forward to reading all opinions.
Cheers!
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Although I'm up for "original" ideas, I just don't think that 5-HT is involved at all, for many many reasons.
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Thanks for the reply, although the first reply means that I didn't express myself properly: we're definitely NOT taking any drugs at all. A month and a half now, with a ton of pills (lion's mane, gingko, magnesium, etc etc), but no drugs, caffeine, etc.
Sleep got much much better. My girlfriend's CEV got better too, although a bout of stress last week brought them back for the night, which means we're not in the clear yet.
I'm still researching.
Possible qEEG / Neuro Feedback medical trial
in MAIN AND GENERAL FORUM
Posted
Aaaannnndd?