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Hallucinogen Persisting Perception Disorder (HPPD) Support Forum


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TheMythos last won the day on May 31 2018

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About TheMythos

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  1. I'm just going to assume you've tried all the usual suspects for anxiety?
  2. Good to hear. We're all probably going to get sick or deal with an illness at some point in our lives (the whole Buddhist aging, sickness, and dying thing)...better to have faced that stuff now to know our strength. I believe you will continue to heal until you no longer think about this disorder and it becomes a footnote in your life.
  3. I won't agree because you aren't a neuroscientist and I don't think you know what you're talking about. I read the entire thread. I play devils advocate and remain skeptical in the name of science and finding answers. For that I'm attacked as somehow denying reality because it's my way of dealing with brain damage...or something. The most plausible theory we have of how psychedelics work in the brain is that they desynchronize certain brain regions, altering the brain waves and magnetic field around the brain. Why couldn't that be off or altered in some way? Why does it have to be cell death caused by glutamate excitotoxicity? You don't get to determine who's a productive member of this forum and who isn't. You've already been banned once for insulting people, so I don't really think you have any leg to stand on like you're this paragon of civility and tolerance on this forum.
  4. It sounds like some strange version of sensorimotor ocd. Could have been there all along, the stress of the trip and no sleep exacerbated it and brought it to the surface.
  5. I think we should all get genetic testing and see if there are any similarities in our genomes. I'm a C667T homozygous for MHTFR. Apparently I have moderate trouble metabolizing folate. If you don't know what that means it can mean neurotransmitter imbalances and less natural antioxidants like glutathione and an increase in homocysteine. In other words, dirty neurons/cells. You can get the test from 23andme and then go to https://www.selfdecode.com which gives you a comprehensive health analysis based on your genes. Also I had something where my 5ht2a receptor is sensitive and it said I could be sensitive to the side effects of SSRIs because of this.
  6. I see images in my head every day and feel like my consciousness and nervous system are being raped. 2 1/2 years off all illicit substances. Quit smoking 6 months ago. Just quit caffeine.
  7. My symptoms tend to come and go based on stress (or with migraines). I used to get snow but now it's gone or I filter it out. My dad sees black static on the wall too and floaters but he doesn't have hppd and it never bothered him. I used to have after images but those went too.
  8. People that have never had to deal with mental problems or brain issues in their life will usually be dismissive because they've never experienced it. "You don't have anything to be sad about" is classic mental health ignorance. You should try to go out and hangout with your friends whenever you get the opportunity though.
  9. I took the Amanita like 11 years ago. You really think I'd be poisoned after all this time? How would uridine help?
  10. I've been thinking of naltrexone... maybe try that first because it shouldn't take as long as keppra to work.
  11. Nobody knows and it doesn't help you in any way to speculate until concrete studies give specific answers and treatment recommendations. Almost every antipsychotic is a 5ht2a antagonist (such as risperidone) and they tend to make a lot of people worse. The best line from that Wiki article is "but the visual system is probably too complex to know for certain." Magnesium is an NMDA (glutamate) antagonist but if magnesium was "the answer" this forum probably wouldn't exist because people would just be like "Oh HPPD? Take some magnesium." I don't know who Dr. Nichols is but he's speculating. I'll leave you with a quote by William James, the father of American psychology: "My experience is what I agree to attend to. Only those items which I notice shape my mind." in The Principles of Psychology, Vol.1
  12. First - stop smoking weed. It isn't helping. Second - it would be beneficial for you to stop researching this and focus on overall health, mental and physical. I know you want it to go away. We all do. A bunch of us researching this on the internet trying to put the pieces of a puzzle together isn't going to bring any answers until some serious funding and medical research takes place. 2 guys in a lab doesn't mean shit until other researchers get on board and do multiple randomized double-blind peer-reviewed studies with sufferers and non-sufferers participating. Third - figure out what supplements may help and try them, along with exercise, healthy diet, meditation, yoga, tai chi and distraction. Try to socialize and do fun stuff even if you don't feel like it. Force yourself into the world. The best thing anyone suffering from HPPD can do is try to get their stress and anxiety down. Most symptoms should start to go with time as long as you live healthy and stay away from drugs. I know it's hard being sober but you don't really have a choice anymore unless you want this to get worse over time.
  13. "First identified in the 1950s by Rita Levi-Montalcini and Stanley Cohen, who later won a Nobel prize for its discovery, NGF has been extensively studied and shown to be one of the primary building blocks of brain health and neuroplasticity. The presence of sufficient NGF enhances cognition by encouraging the growth and branching of axons, the long threadlike nerve cell extensions along which impulses are conducted; insufficient NGF impairs cognition by leading to neuronal decay and the eventual death of nerve cells. The more axons that exist and the better their health, the more rapidly and efficiently impulses can be transmitted and the better the cognition and overall brain function." Glutamate excitotoxicity isn't the only thing that causes cell death. If HPPD is neuronal death (big if) we need to look at NGF and BDNF since exercise helps so many people as well as Lion's Mane and other adaptogenic, nootropic herbs. The reason some may have been helped with LONG-TERM antidepressant use is because of the neuroplasticity that SSRIs promote and the reduced glutamate signaling over months of 5ht2a downregulation. SSRIs help attenuate stress within a month. Once someone's stress response lowers they feel better and feeling better creates a positive snowball effect where they can adequately eliminate certain stressors that are making them worse, improving brain function and nervous system signaling.
  14. Can someone a little more knowledgeable explain why you would choose Keppra over it's analogue Briviact when Briviact has less side effects and a 20 fold more affinity for the same MoA?
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