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VisualDude

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VisualDude last won the day on February 13

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About VisualDude

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  1. Its hard to know why things are quiet. I myself am here only occasionally in recent years. Like Jay mentioned, it can be helpful to talk with others who suffer the same things - to those who haven't, some of this stuff in incomprehensible to them. If one can keep their head cool and patiently look at what has helped others (which is quite varied), then one can try things that might help. ( No bleach though ) Perhaps Covid19 and the hatred that has been stirred up between people in recent years ... there isn't as much participation in forums dedicated to actually help. On some for
  2. Since the topic of anxiety and HPPD is perhaps the most frequent, it might be helpful to understand some of the processes going on. It seems true that most people with HPPD have anxiety issues. A rare few have no anxiety with their HPPD. Then there are plenty that find anxiety aggravates HPPD … and controlling anxiety alleviates HPPD at least to some degree. Most are familiar with the amygdala and one’s fight/fright response. But more is involved. To start with, many brain functions are compartmentalized. Some functions are primarily in one physical location. Others are inter-w
  3. Since you got it from a "bad trip with visual hallucinogens", that is defining point of HPPD that is not VSS. But really, the diagnosing belongs to doctors who understand the disorder(s) --- good luck finding one. Also, to some degree the point is mute because there is no established medical protocol to treat either. You just have to try things with doctor(s) who are willing to help. Finally, people don't become schizophrenic or die from HPPD or VSS. It can just be very unpleasant and for some, interfere with work. The visual problems are 'illusions' not hallucinations.
  4. Glad it is helping you Trazodone affects so many receptors, its hard to know what is helping (other than it overall 'quiets' many circuits) See https://en.wikipedia.org/wiki/Trazodone#Pharmacology It states in this publication http://www.facesofhppd.com/uploads/1/0/8/8/10884075/hallucinogen_persisting_perception_disorder_and_flashbacks_2013.pdf "Abraham & Duffy (1996) hypothesized that HPPD is a disinhibition of visual processing related to a loss of 5-HT-receptors on inhibitory interneurons." The 5-HT receptors that are inhibitory are the 5-HT1 and 5-HT5 receptor ty
  5. It is important that neurotoxicity, including that from hyperactivity (regardless of being glutamate or other neurotransmitters), involves the accumulative effect - that is, total toxic burden at the time. The visual from LSD are attributed largely to one class of serotonin receptor which caused visual processing to go on overdrive, not to mention other effects such as 'ego-loss'. So while LSD is less damaging than crack and meth, it still can add to the burden. MDMA is essentially an immediate acting SSRI without sedating effects. So the increased serotonin can be toxic. For example,
  6. Love your positive attitude ! It is also true that many things that have been discussed are buried in the past, often not easily seen.
  7. When people post as he did, it is best to not get too caught up in it because the manor was unreasonable. Why? Troll? Egomania? Just over exuberant? Who knows. But his 'style' is not unlike people popping onto forums and posting, "If you just pray to Jesus sincerely enough, he will miraculously cure you". The reception to this is negative for obvious reasons. [ And if one is into the Bible, it grossly misrepresents the whole situation we live in now ]. The whole topic of controlling anxiety has been discussed and will be discussed and will remain an important t
  8. This is true of most diseases and disorders. Why don't you (and other members) let the doctors do the diagnosing - they are the ones who defined the disorder. Read more posts. Some have no anxiety. Some are actually glad to have the visual anomalies. I personally know one on the DPSelfHelp forum. You think you know all Native Americans? So your personal experience make you knowledgeable about everyone's personal experience? n=1 is a very small sample for statistics Clearly you are very impressionable. Perhaps you have learned
  9. Not everything wrong is a conspiracy. Habits too can be hard to break. This study indicates the old suggested dosage being 400-600 IU/day was based on preventing rickets. This study also puts the normal daily dose "around 8000 IU for young adults and thereafter" https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5541280/pdf/jpmph-50-4-278.pdf Because D toxicity can be nasty and take 6 months for levels to drop to normal, one needs to monitor high dosage taken long term. D absorbs in adipose so heavy people may not show good serum blood levels for some time ... then if they start losing weig
  10. Would like to see data on that one
  11. I've taken 15,000 iu / day for a couple weeks but decided to back down to 10k. At 6 months of 10k/day, and prior to any osteoporosis meds, D3 (D 25-Hydroxy) was 82 and the active form (D 1,25 DIHY) was 62. These are both roughly 80% into the range. A couple years later with the meds Forteo and Prolia at the same time, D3 was 52 and the active form 72. Forteo is a synthetic portion of parathyroid hormone (PTH). Both work by stimulating the conversion of D3 to its active form. Am told this happens in the bones and the kidneys. But don't know about in the brain, although both forms
  12. Good article. A note about dosage. The maximum daily dose long term is 4,000 iu without blood test monitoring. [ Ref missing right now but that is for all adults worldwide ]. To put this in perspective, if you are in the sun a lot, the the skin will make up to 10,000 iu a day. I post this because lots of articles about the danger of Vitamin D as well as many doctors feeling one only needs 200 iu/day. If I find the ref I'll post it but one can be safe with the above dose indefinitely. I am currently taking 10,000 iu for 3 years now (with blood tests). Since am on multiple meds f
  13. I've tried Propranolol with minimum helpful effects. However sexual dysfunction was worse. To see if the opposite would help SD, tried Salbutamol (pill form of albuterol) which is adrenergic β2 receptor agonist. Was concerned about trying since common side effects are tremor, anxiety, headache, muscle cramps, palpitations and sleep problems. Already have these problems but tried it anyway. It actually helps vision (especially feeling disconnected) and helps RLS and anxiety and SD. Tremor a little worse but nothing of concern. Also helps breathing as I have asthma and this is an asth
  14. For what its worth (and in theme of the thread), the use of testosterone helps depression. It is well know that low T causes a depressiveness. This article is about using high dose testosterone for major depression. https://www.ncbi.nlm.nih.gov/pubmed/30427999 "Testosterone treatment appears to be effective and efficacious in reducing depressive symptoms in men, particularly when higher-dosage regimens were applied in carefully selected samples."
  15. Interesting that selegeline helps you. I've used it and it was helpful. Problem was doctor moved and other docs were afraid. By the time I tried it again, it wasn't all that helpful. I have RLS bad. Doesn't bother me but bugs others including my wife. First time using selegeline, it stopped and the change was so significant that at night my wife kept worrying I had died since I had never been so still. Later docs tried Abilify and the restlessness came back (had been much quieter for months after selegeline). Trying selegeline again had no effect on RLS. Typical with HPPD (and
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