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Found 15 results

  1. long story short i got hppd back in 2019 from using lsd once. went through all the motions and managed to beat it and got used to it after 6 months. stupidly done cocaine 3-4 times over 3 years and noticed that i mightve made it ever so slightly worse whereas sometimes it didnt affect me. recently done coke twice in october and now my visual snow is on every single surface, i cant unsee it. auditory hallucinations have started as well, its not schizo as i have spoken with others who experience the exact same thing as me... brain chatter at night and in the morning and ill usually always have a song going through my head all throughout the day. its getting unbearable, for the last 3 weeks ive thought about suicide everyday... ive reached out to specialists in the uk but havent heard back... worried ive fucked my life up. I got through this the first time and was enjoying every aspect of life and now im back in this hole and dont see a way out. pls help
  2. Hi, I am new to this website, however, I have been lurking around for some time as just a spectator. Has anyone tried VA or Memantine to treat late stage HPPD symptoms. I've been improving steadily for the better part of 4 months now after being in a free fall for six, tinnitus, light sensitivity, and balance issues are all gone. However, the visual snow and occasional hypochondriac attack still occur. Mostly I've been running off my issues and it has put me in good health, for medications I have been on 50mg of Imipramine in addition to 200mg of Lamotrigine. This combination works great, once you get past the original side effects. I also take Huperazine A in order to combat the cholinergic antagonism displayed by the Imipramine and it keeps the memory problems at bay (if anything it has been improving my cognition). I've been considering in trying out VA (Anticonvulsant) and Memantine (NMDAR antagonist) in the treatment of my condition. The memantine is already on the way, however I have yet to act on the VA. My question is as to whether VA is worth it? I've been reading up on it and have been seeing pretty mixed things on the drug. Ranging from psychotic episodes, to it not doing anything, to creating perfect pitch. Sounds like there's a lot of risk involved in it. As for Memantine, one of the theories that I've seen floated, especially that floated on the r/HPPD subreddit, is that there is some degree of Glutamergic dysfunction within neurons. Would Memantine optimize Glutamergic Transmission or would it just make it worse?
  3. I cut drug use one month ago and my visuals everyday are sligthly worst is that normal guys?
  4. Im wondering on your experience with oxycodone and hppd specifically your visuals.
  5. Hello guys, it's been a year since i had the same problem as you. Always exactly 1 year I took 150ug of LSD and had a terrible bad trip that traumatized me and left me sequels. I had flashbacks and I missed a lot because of it. Distorted visions in my peripheral field. All this was cured with antipsychotics, especially risperidone (I can not remember the dosage). But what it took to be cured was the emotional sequel that caused me. Psychedelic experiences transform you radically and unfortunately if you are not prepared for them it may take a long time for you to get back on track and I confess that I am not yet 100% healed of this trauma. But what I can say to you is that the worst of all this is not the visions, but the horrible thoughts that go through our heads thanks to the anxiety that causes us. And what I want to know about this post is just that. What are the thoughts that torment you because of this anxiety? Write them in the comments and we'll help each other by talking about them. For example, I used to think all the time that I was getting schizophrenic, that there would be some outbreak, over time this evolved into existential crises where I thought my soul was lost in space time and I was not living reality, just watching it (depersonalization and derealization helped in this).
  6. i was wondering what were your baclofen experiences were as i was perscribed it recently
  7. Im wondering on your experience with oxycodone and hppd specifically your visuals.
  8. i was wondering what were your baclofen experiences were as i was perscribed it recently
  9. Hello i was recently prescribed soma (carisoprodol) for my back pain, i was wondering how it affected your visuals as i'm contemplating on taking it. A little background is that my hppd is super mild, i've only had it for around a month, i barely get after images and i get slight tracers which are not noticeable too much during the day but get slightly worse at night when bright objects move past my field of view eg. a phone screen. Also i can take benzodiazepines like diazepam and gabaergics like gabapentin without a change in any visuals. thank you for reading and i'm looking forward to your responses(please don't be negative). :)
  10. Hello i was recently prescribed soma (carisoprodol) for my back pain, i was wondering how it affected your visuals as i'm contemplating on taking it. A little background is that my hppd is super mild, i've only had it for around a month, i barely get after images and i get slight tracers which are not noticeable too much during the day but get slightly worse at night when bright objects move past my field of view eg. a phone screen. Also i can take benzodiazepines like diazepam and gabaergics like gabapentin without a change in any visuals. thank you for reading and i'm looking forward to your responses(please don't be negative). :)
  11. A few nights ago i decided to take mdma idiotically because my symptoms were so much better after 4 months of abstaining from drugs, well during the trip and afterwards i can say i believe i know whats it like to have the full 9 yards of hppd. During the trip it was horrifying, faces everywhere on my floor on my dogs fur on my wall, my feet were itchy i could feel the anxiety and paranoia but i think the mdma helped me manage to stay happy. The next day i learned of all the severity of symptoms that other people have that i dont, my ghosting was not just only above like it usually is, but it was below and side by side, 4x as worse as what it usually was. When people talk about starbursting, i used to think that was normal, but no i saw what it really was like, and i saw afterimages of literally everything. My visual snow was creating flying cars or weird hallucinations that i couldnt completely make out, my anxiety had been almost the same as it was before. Thankfully 4 days later it seems as if im back to the baseline as to where it was before. Guys, mdma used to be my drug, i used to do it every day before work but now i cant stomach it. So my message to you is, if you have hppd, DRUGS ARE NOT WORTH IT.
  12. Hey! My name is Sander, I'm 15 years old, I'm from Amsterdam and I've been suffering from HPPD for excactly a year now. I have this party coming up, and I was wondering, How big are the chances ofmy HPPD worsening if I do coke or get drunk? Thanks!
  13. If no one minds, I'd like to create and maintain this thread to compile articles, book excerpts, studies, quotes, etc. on orthomolecular approaches to treating/managing and reversing neuro-psychiatric illnesses, HPPD being one (if it's a problem, no worries, it can be deleted or whatever). When I first started getting HPPD symptoms (which were much less severe than now), I started looking into things like organic foods (I definitely recommend the documentary Genetic Roulette, as well as the book Seeds of Deception, for more information), followed by alternative and naturopathic medicine. Nowadays, I'm open to anything, including the traditional, Western allopathic model to medicine. Nonetheless, personally, I believe the orthomolecular approach holds the greatest promise as it is (1) science based (unlike some other models), and (2) contains potentially hundreds of thousands of cases of people being cured of their ailments, which, interestingly include ailments such as psychosis and Schizophrenia. To begin this, I'd like to share some excerpts from an article on the thyroid (hormone)/oxidative metabolism-"insanity" connection written by Dr. Ray Peat; though I'm a fan of Dr. Peat, there are many others that have fantastic information, such as (but not limited to): Dr. Abram Hoffer, Dr. Albert Szent-Gyorgi, Dr. Linus Pauling, and Dr. Roger J. Williams. Thyroid, insomnia, and the insanities: Commonalities in disease SOME FACTORS IN STRESS, INSOMNIA AND THE BRAIN SYNDROMES: "Everyone is familiar with the problem of defining insanity, in the case of people who plead innocent by reason of insanity. The official definition of insanity in criminal law is “the inability to tell right from wrong.” Obviously, that can’t be generalized to everyday life, because any sane person realizes that certainty is impossible, and that most situations, including elections, offer you at best the choice of “the lesser of two evils,” or the opportunity to “do the right thing,” and to “throw your vote away.” People who persist in doing what they know is really right are “eccentric,” in the sense that they don’t adapt to society’s norms. In a society that chooses to destroy ecosystems, rather than adapting to them, the question of sanity should be an everyday political issue." "Looking for general physiological problems behind the various symptoms is very different from the practice of classifying the insanities according to their symptoms and the hypothetical “brain chemicals” that are believed to “cause the symptoms.” The fact that some patients hallucinate caused many psychiatrists to believe that hallucinogenic chemicals, interfering with nerve transmitter substances such as dopamine or serotonin, were going to provide insight into psychotic states. The dopamine excess (or serotonin deficiency) theories developed at a time when only a few “transmitter substances” were known, and when they were thought to act as very specific on/off nerve switches, rather than as links in metabolic networks. The drug industry helps to keep those ideas alive." "A particular drug has many effects other than those that are commonly recognized as its “mechanism of action,” but when an “antidepressant” or a “tranquilizer” or a “serotonin reuptake inhibitor” alleviates a particular condition, some people argue that the condition must have been caused by the “specific chemistry” that the drug is thought to affect. Because of the computer metaphor for the brain, these effects are commonly thought to be primarily in the synapses, the membranes, and the transmitter chemicals." "The brain, just like any organ or tissue, is an energy-producing metabolic system, and its oxidative metabolism is extremely intense, and it is more dependent on oxygen for continuous normal functioning than any other organ. Without oxygen, its characteristic functioning (consciousness) stops instantly (when blood flow stops, blindness begins in about three seconds, and other responses stop after a few more seconds). The concentration of ATP, which is called the cellular energy molecule, doesn’t decrease immediately. Nothing detectable happens to the “neurotransmitters, synapses, or membrane structures” in this short period; consciousness is a metabolic process that, in the computer metaphor, would be the flow of electrons itself, under the influence of an electromotive force, a complex but continuous sort of electromagnetic field. The computer metaphor would seem to have little to offer for understanding the brain." "Although it is common to speak of sleep and hibernation as variations on the theme of economizing on energy expenditure, I suspect that nocturnal sleep has the special function of minimizing the stress of darkness itself, and that it has subsidiary functions, including its now well confirmed role in the consolidation and organization of memory. This view of sleep is consistent with observations that disturbed sleep is associated with obesity, and that the torpor-hibernation chemical, serotonin, powerfully interferes with learning. Babies spend most of their time sleeping, and during life the amount of time spent sleeping decreases, with nightly sleeping time decreasing by about half an hour per decade after middle age. Babies have an extremely high metabolic rate and a stable temperature. With age the metabolic rate progressively declines, and as a result the ability to maintain an adequate body temperature tends to decrease with aging. (The simple fact that body temperature regulates all organic functions, including brain waves, is habitually overlooked. The actions of a drug on brain waves, for example, may be mediated by its effects on body temperature, but this wouldn’t be very interesting to pharmacologists looking for “transmitter-specific” drugs.) Torpor is the opposite of restful sleep, and with aging, depression, hypothyroidism, and a variety of brain syndromes, sleep tends toward the hypothermic torpor. An individual cell behaves analogously to the whole person. A baby’s “high energy resting state” is paralleled by the stable condition of a cell that is abundantly charged with energy; ATP and carbon dioxide are at high levels in these cells. Progesterone’s effects on nerve cells include favoring the high energy resting state, and this is closely involved in progesterone’s “thermogenic” effect, in which it raises the temperature set-point. The basal metabolic rate, which is mainly governed by thyroid, roughly corresponds to the average body temperature. However, in hypothyroidism, there is an adaptive increase in the activity of the sympathetic nervous system, producing more adrenalin, which helps to maintain body temperature by causing vasoconstriction in the skin. In aging, menopause, and various stressful conditions, the increased adrenalin (and the increased cortisol production which is produced by excess adrenalin) causes a tendency to wake more easily, and to have less restful sleep. While the early morning body temperature will sometimes be low in hypothyroidism, I have found many exceptions to this. In protein deficiency, sodium deficiency, in menopause with flushing symptoms, and in both phases of the manic depression cycle, and in some schizophrenics, the morning temperature is high, corresponding to very high levels of adrenalin and cortisol. Taking the temperature before and after breakfast will show a reduction of temperature, the opposite of what occurs in simple hypothyroidism, because raising the blood sugar permits the adrenalin and cortisol to fall." "Therapies that have been successful in treating “schizophrenia” include penicillin, sleep therapy, hyperbaric oxygen, carbon dioxide therapy, thyroid, acetazolamide, lithium and vitamins. These all make fundamental contributions to the restoration of biological energy. Antibiotics, for example, lower endotoxin formation in the intestine, protect against the induction by endotoxin of serotonin, histamine, estrogen, and cortisol. Acetazolamide causes the tissues to retain carbon dioxide, and increased carbon dioxide acidifies cells, preventing serotonin secretion."
  14. This post has been promoted to an article
  15. I found this drug online called Kanna(sceletium tortuosum). It apparently mixes well with weed and is supposed to just be very calming. Some websites are saying it could be considered an ssri which probably isn't a good idea to do but I'm not sure. I have'nt smoked anything but weed in while and I'm not sure I should try new drugs anymore because my hppd is calming down. Have you guys heard anything about this stuff? http://azarius.net/smartshop/psychedelics/psychedelic_herbs/sceletium_tortuosum_kanna/
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