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BigPapaChakra last won the day on May 12 2014

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  1. I do/did, but not at all related to hydrocephalus or anything similar to that. I've had a bunch of very bad concussions, cracked my head open, and had other head injuries in which I never went to the hospital/doctor, but were just as bad as other ones that caused concussions. I also have an upper-cervical disc bulge and have had some back and neck injuries.
  2. Peptide regulation of ageing Introduction This is a site from some of the people who created these peptide bioregulators. There is a lot of evidence behind them, but much of it is in Russian or other Eastern European languages, along with various patents. Below are some of the graphs/info I found interesting (it's a very small excerpt, though, lol): Taking into consideration a reliable biological activity of peptides we found it reasonable to study the effect of regulatory peptides in monkeys. Restoration of the melatonin level up to normal following the administration of the peptide preparation to old monkeys was among our significant achievements. Fig. 21. The peptide effect on melatonin production in monkeys of different age. The same old monkeys revealed a restoration to normal indices of a daily rhythm of secretion of the main hormone of adrenal gland – cortisol (Fig. 22). Fig. 22. The peptide effect on cortisol production in monkeys of different age (in the morning and in the evening). Table 2 Peptide preparation effect on mortality rate in elderly and old patients Group of patients Indices Control (administration of polyvitamins) Administration of the pineal gland preparation Administration of the complex of thymus and pineal gland preparations Elderly people (60-74 years) Initial mean age, years 69.3 ± 2.2 71.1 ± 1.4 No studies Mortality rate in the course of 8 years, % 13.6 8.5* Mortality rate in the course of 12 years, % 44.1 22.3* Old people (75-89 years) Initial mean age, years 80.2 ± 1.6 81.5 ± 2.1 82.1 ± 2.3 Mortality rate in the course of 6 years, % 81.8 45.8* 33.3* * p<0.05 compared to the control Fig. 23. Effect of thymus preparation on metabolism in elderly patients (60-74 y.o.). Fig. 24. Dynamics of reaction of blast-transformation of lymphocytes with phytohaemagglutinin index in elderly patients in 3 years after 6 courses of peptide preparations. Fig. 25. ARD incidence in elderly and old people treated with thymus preparation. Fig. 26. Effect of the pineal gland preparation on melatonin level in elderly people.
  3. Jacob Liberman has a book on light and vision training, too. There are downloads for it online. His research on light, eyes, and the brain led him to look into meditation and neurofeedback and he has become rather spiritual, so there is a lot of talk of spirituality in his book, but it has sound information. He also recommends a product in his book that is affordable and can be hooked up to your PC (I believe) that trains vision, particularly the skill of vision, not only visual acuity. Z-health has a vision training product, too, that is grounded in research. Same with the Feldenkrais method if I'm correct. Anyhow, what I've learned from z-health is that many visual or vestibular problems are BOTH visual AND vestibular problems - so you'll get optimal results if you train vision+the vestibular apparatus at the same time. It's perfectly fine to train your vision and vestibular apparatus at home, and I've begun doing so, but I'm not sure this should be highly recommended without prior research and guidance by a clinician, especially in someone with severe neurological/psychiatric issues. Many times different brain regions "wake up" and can cause temporarily negative reactions - even from simple eye movements. I've experienced this myself, and have often seen it discussed in interactive metronome and z-health groups.
  4. Reminds me of syntonic optometry. A lot of people practicing optometry that utilize syntonics have largely reversed neuro-psychiatric conditions in some people with the use of light, vision training, psycho-spiritual stimulation by impacting brain regions, etc. Check out some of these resources: College of Syntonic Optometry Jacob Liberman, O.D., Ph.D., D.Sc. Eyelights Therapy
  5. I had very similar reactions initially. I first did a NeurOptimal neurofeedback session, and during it I felt very peaceful and extremely happy. The rest of the day I was extremely in tune with things. The next day I was in a state of constant fear and panic beyond what I would even normally feel. I basically just laid down the entire day fighting back the sensations of a nervous breakdown. The following days things got better. The woman was very kind and kept insisting I come in for a quick session for less or no money so she can check on me and make sure I'm not suffering. Anyhow, this is what led me to Dr. Lori-Russell Chapin. I did SMR-neurofeedback (sensory motor rhythm). During the sessions I literally felt a state of orgasmic bliss wash over my body. I had tactile sensations in my face/behind my eyes akin to what I've experienced with marijuana edibles after a long "tolerance break". The next day I felt more anxious, again, but much less so than my initial experience with NeurOptimal. After my second SMR-session, I felt even better and even started writing music/lyrics and studying again; the next day I felt more anxious, but I was also more capable of handling the anxiety, and with proceeding days I felt better than my HPPD-baseline. Anyhow, Dr. LRC was about 3 hours away from my home, charged really large amounts of money, and her personality didn't mesh with mine. I decided to stop seeing her after these sessions and get a nIR HEG device from along with the "LIFE game" to open focus, closed focus, and the ability to intentionally switch between open and closed focus. I haven't really used it because I've found that other issues, such as Lyme and biotoxins, are increasing my HPPD symptoms, and it doesn't seem plausible that neurofeedback will address those problems directly, and that any negative experiences I could potentially experience with NFB is from these infections. So, I plan on addressing these issues, along with diet, first, and then working my way towards increasingly immense amounts of NFB, particularly nIR HEG and TLC7a NFB. I have been doing biofeedback, though, for about 5-10 minutes a day, and will soon be adding Tai Chi into the mix.
  6. Oh, by the way, there is a yahoo-neurofeedback group, and both Pete and Douglas post there. Douglas has made posts there that rival the information he posts on his own websites, and truthfully, the man is a genius. He's found ways to take people who had adverse reactions to neurofeedback, or who were "non-responders", and get them to respond favorably. Not only this, he's found ways to combine tDCS+neurofeedback to enhance one's neurofeedback session, and additionally, how to do acupressure/acupuncture at specific points alongside TAG-Sync and meditation to directly alter the Default Mode Network and get people to have very therapeutic sessions in short amounts of time. Anyhow, I posted on the yahoo group long ago about my problems, my research, etc. A man with similar interests started responding to me, and coincidentally, his interest in neuro and biofeedback was caused by substance abuse-induced brain damage, including PCP (which is one of the compounds I attribute my HPPD to). He has used the TLC7a approach to neurofeedback and stated that it completely changed his life, whereas other forms of neurofeedback made him more anxious, dissociated, etc. furthermore, he has even better cognition now than before many of his problems, and has thus started to use nIR HEG concurrently with the TLC7a approach. Gonna re-start my nIR HEG training very soon. Can't wait.
  7. Jess, now you have me wanting to converse with you even more. I just posted on your thread created about your video. Anyhow, biofeedback and neurofeedback is something I've been experimenting with and researching for the past couple months, and may even get certified in. Let me first say that not all neurofeedback is the same, yet many people think it is, or at least think that it's okay to "up train" or "down train" certain frequencies in certain regions of the brain, and that is all it is. This is false. Do you know what protocol your neurofeedback practitioner is using? Do you know what technology or software they're running? Are they asking you to do anything while you have electrodes on? Recently I did some work with a professor at a university in my state and a leading researcher of the brain research center out of that university. Her name is Dr. Lori-Russell Chapin. Her and her husband wrote a book on neurofeedback, and are 2 of 4 people utilizing Theta-Alpha-Gamma Synchrony training in the U.S. (perhaps the world?) - one of the most revolutionary protocols in not only neurofeedback, but neuromodulation in general. I also worked with one other neurofeedback clinician prior to this, and am going to consult with one more who in fact has invented some new technology and protocols himself. One thing I'd like to point out is that, when reading the research behind neurofeedback, temporary adverse reactions are common in people with deeply rooted problems. I have experienced these first hand. Some with PTSD even had flashbacks and hallucinations, but with further training, not only were cured, but had better cognition than their pre-PTSD baseline. Anyhow, QEEG-driven neurofeedback is sub-optimal. TLC7-assessment directed neurofeedback is where real results come from, and after that, one can do some extensive protocols designed for glial cell functioning, neurogenesis, etc. TLC7A's are an extensive look into your brain functioning compared to your own brain, whereas a QEEG compares your brain to a database of "normal brains". Changing your neurofeedback practitioner may be needed, but truthfully, saving up the money and running a TLC7A and doing personalized neurofeedback with consultations with Peter Van Deusen or Douglas Daily would be optimal. Anyhow, I recommend looking into: - a TLC7 assessment, done at home, and TLC7A-directed individualized neurofeedback, which minimizes negative effects and maximizes results, far beyon what any QEEG-driven neurofeedback could accomplish - TAG-Sync, or Theta-Alpha-Gamma Synchrony, which causes massive neuronal changes in the Default Mode Network among other areas, similar to what occurs in people who have meditated daily for decades, and affects the neuro-physiological causes of negative emotions and senses directly - BiPolar Montage neurofeedback - Ultra/Extremely Low Frequency Neurofeedback - Slow Cortical Potential Neurofeedback - near or passive infrared hemoencephalography (pIR/nIR HEG) neurofeedback, which directly treats hypofrontality, something that largely occurs in schizophrenia For further reading: TAG-Sync (Douglas has multiple sites, this is the least extensive explanation of TAG-Sync, but is a good intro) Pete Van Deusen/Brain-Trainer TAG-Sync experiences on longecity (the creator of this thread has spoken to me frequently in the past, all I can say is, in terms of his language online, he seems like a completely evolved, extremely efficiently functioning individual, 100% due to his use of TAG-Sync, and he was a healthy individual prior to using it...) I'd really like to describe some of these things, and others, to you in more depth.
  8. Nice video, Jess! It's courageous of you to put yourself out in that manner to make your experience with ibogaine and your proceeding state of being known to the public. I truly feel for you. In our brief conversations on here, you've across as a kind person, and you appear to be young and attractive - you don't deserve to feel the way you do There's a bunch I'd like to say, but knowing myself I'll end up rambling. That being said, I've been doing a lot of contemplating, experimentation, research, conversing with researchers/clinicians, etc. lately, and believe that you, as well as anyone else here suffering from severe HPPD/co-morbidities, could benefit from a lot of different modalities. Considering the neurological/sensory disturbances you described, what you stated the doctors thought could/did happen, your previous experiences with certain meds, etc. some things come to mind for effective treatments. I was planning on making an update on here due to my recent absence, in which I can describe some of the things I'm alluding to. It'd be awesome if you made an HPPD channel. If you're interested in what I stated above, or ever need anything, feel free to message me
  9. Extended fasting has cured treatment resistant Schizophrenia in Russia; but they fast for like 30-60 days with just mineral water and epsom salt baths, then after slowly add foods with progressing weeks, and there after fast like once a month for 3-5 days, then after awhile once every other month, and so on. I plan on trying this in the future. I'm going to be going back to ketosis, but with an emphais on saturated and mono-unsaturated fats in particular; I'll post about why it would be wise to avoid unsaturated fats (including o3's), especially in ketosis, when I make an update post. I'd look into Dr. Broda Barnes' material, too, along with Dr. Jan Kwasniewski. Coconut oil and milk, mct oil, caprylic acid (oil), leucine and lysine, ketone salts and esters, 2-deoxy glucose, butanediol, and other compounds promote ketogenesis despite carb intake. All those, plus fasting, can be used to generate beta hydroxybutyrate without eating like 0-30g/carbs/day.
  10. Just try CES, it's cheape, safer, can be done at home, more versatile, and it works. I've posted studies about it before, in which it caused severely psychotic, violent inpatients drastic (positive) changes in their behavior, some of which were discharged, and many of which no longer needed to be tranquilized and needed reductions in anti-psychotics. I've started it up again. One of the only things I've found effective, and I'm using a very old model (made like 10 years ago); there are new ones with more effective wave-forms, along with some choice in the brain frequencies you want to produce. The one I have only allows me to do 0.5-1.0hz or 100hz; I'm looking into others that allow (in addition to the previous two frequencies) the Schumann Resonance, sensory motor rhythm, alpha, and theta frequencies. I also recently found out about The Walsh Institute and some others, which does some amazing nutrient therapies and their focus is on people with severe psychiatric problems; they even do studies on death row inmates with extremely violent histories, and get them to change. They want to cause a new era of psychiatry based off of extensions of the work of old orthomolecular psychiatrists such as Dr. Pfieffer, and cause a drastic reduction in school shootings and murders and the like via getting peoples neuro-chemistry balanced. I'll post about this in depth elsewhere.
  11. Just found out curcumin may increase NMDA NR2A sub-unit density AND protect against NMDA-induced excitotoxicity. There are supposedly some more potent forms of curcumin becoming available, more potent than even BCM-95 and similar brands with turmeric fats attached. Some also do IM or IV injections of curcumin. Based off of some of the studies I've looked at, as with most things the benefits accrue with time as it will eventually increase the density these NMDA subtype receptors, decrease HS CRP, and more. The benefits should be anything but noticeable before a period of at least a month, and even then it may take some more time and/or larger doses. It also appears to increase (or protect?) hippocampal progenitor cell proliferation, much akin to NSI-189. Spirulina has also been used in clinical trials (that I posted at the HPPD stack thread) to protect hippocampal progenitor cells produced from some proprietary supplement formula that really has "natural NSI" written all over it. It'd be interesting to combine NSI+spirulina+curcumin. Or potentially curcumin+MgT and other NMDA-agonizing nutrients/drugs/compounds. May give this a try after using a few grams of pregnenolone, lol.
  12. I've stopped everything and have been entirely sober for well over a year, I don't know exactly how long. For the first months after HPPD, I continued to use cannabis, alcohol, and occasionally benzos. When I smoked, I would be catapulted into a full-blown panic attack with the sensation/experience some describe as "impending doom". Additionally, all my visual symptoms were greatly increased, including visual snow. Also, I'd get a mix between a traditional migraine and an occipital/visual migraine. For instance, I'd get flashes of light and these big blobs/orbs of color which some experience prior to getting a migraine, or experience solely if they have occipital migraines. Yet I'd get these visual symptoms along with the migraine. Yet, if I was drunk, I could smoke a small amount. If I was on a benzo, I could smoke as if I didn't have HPPD. Yet since my HPPD has gotten worse, if I had ever chosen to drink and take a benzo, I'd still never smoke as it carries too large of a risk. The experience of being drunk has also been altered. Believe it or not, throughout my period of substance abuse, I never experienced a hang over, and only once felt the need to vomit (and that occasion included a lot of different things). Yet, if I were to take up drinking again, I could assure you that I'd end up getting hang overs and I'd probably vomit occasionally. At probably post-HPPD months 4-6, I went through a period where I drank a lot with my fiance (then my girlfriend, who also at the time was not sober). I could still handle my alcohol, but it took effort. I also started to become dizzy and experience vertigo after a certain amount, whereas before I NEVER experienced this no matter what amount of alcohol I ingested. For the first month or two after HPPD, I would take benzos at concerts to replace my use of substances such as LSD, MDMA, MDA, and Ketamine. These would actually be good experiences despite a relatively high dose (for one without a large tolerance). My visual snow, oddly enough, would decrease. I of course had no anxiety whatsoever, and I had a lack of dissociation. I'd also have great recollection of the prior night, despite the fact that when one abuses benzos they normally don't have much if any recollection of the prior day/night. As I said, I no longer abuse ANY substance whatsoever, but the fact that I have had such enjoyable, controllable, HPPD-free days/nights on benzos has always had allure to me, and that's not good with an addictive personality. Despite that, I've only taken a total of 1 etizolam out of the 20 I originally bought sometime around March, lol. After studying Dr. Selye's research on stress, I definitely see all sources of stress as interconnected. All compounds have direct effects outside of the General Adaptation Syndrome that can cause "stress", thus I see substance abuse, even lightly recreational abuse, inherently stressful. Therefore, I'd also like to state that other forms of stress, such as sleep deprivation or arguments and so forth, also have a much greater impact on me. They increase every HPPD symptom in me, including visual symptoms. Also, when I first wake up any day outside of days that I have "perfect sleep" (as compared to my typical sleep), I'm extremely "tripped out". I'm very dissociated, much more groggy and fatigued upon waking, and have trouble separating my dreams from reality for the first some odd minutes of the day. I'm sure other people have experienced that after vivid or lucid dreams, but this is now a daily occurrence. Of course, I can discern my dreams from reality, but every night I have near lucid dreams and wake up thinking "Did that happen yesterday? Oh, nevermind, lol, that was just another crazy dream." Even meditation impacts me more, and sometimes it worries me due to the whole "kundalini syndrome" thing.
  13. Really interesting that you found that, because just about 2 days ago I found out about the researcher who discovered Nogo-A: Definitely some fascinating information!
  14. Wow, nice find! I've never even heard about these but there is a good post on them, and on a bunch of psychedelic/meditation/etc. boards people have been using them and posting about them for a few years. They appear to be a potent acetylcholinesterase inhibitor, along with decreasing serotonin turnover and excretion (in the urine (so this means increased serotonin, too?)). It's also been shown to enhance myelination of the brain. I believe they protect from glutamate toxicity, too. I may have to try these since they modulate the neurotransmitters/receptors I think may be low/hypofunctional in my particular brain. Thanks!
  15. I would imagine it has something to do with trauma in a sense. I've had multiple, pretty severe concussions and head injuries, and that predisposes one to toxin accumulation in the brain via some mechanism that is not coming to mind at the moment. Yet, there are studies in which rodents when put through a very stressful event early in their life were less resistant to stress down the road, whether it was mental/emotional, or a toxin. So, for us HPPDer's, perhaps the events leading up to acquiring it/co-morbid disorders were akin to stressful events in those experiments and now we're merely less resistant to most things. I know I'm more sensitive to other things, too, for instance, bright light, loud sounds, rapid changes of direction (which can become dizzying now), and more.